Medications

Antibiotics in Respiratory Disease

Core oral and intravenous antibiotics in respiratory infection — with the physiotherapy timing and adverse-effect points that matter.

Primarily for physiotherapists & allied health professionals
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Authorship & review
Dr Sean James Ledger, BSc Physio (Hons) MSc PhD FHEA
Director and Principal Physiotherapist
Ahpra registration PHY0002298174
Version
1.2
Last updated
16 August 2026
Next review
16 August 2027
Every guide on this site is reviewed at least once a year, and sooner when the evidence changes.
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In plain language

Antibiotics treat bacterial chest infections and exacerbations. This page lists the core oral and IV agents, their typical uses and the points a physiotherapist should know — including when clearance is best timed and which agents can cause bronchospasm. Inhaled antibiotics used long-term in chronic infection are covered under nebulised therapy.

Antibiotics treat the bacterial chest infections that drive many respiratory exacerbations.1 The choice depends on the likely organism, the severity and setting, any allergies, and local resistance patterns — guided in Australia by Therapeutic Guidelines: Antibiotic.2 For physiotherapy the key point is that antibiotics and airway clearance work together: clearing secretions helps the drug reach the airways, and starting a course is often the trigger to step up clearance during an exacerbation. Inhaled and nebulised antibiotics are also used for chronic infection in bronchiectasis and cystic fibrosis, and are always given after airway clearance. The table below summarises the agents commonly seen in cardiorespiratory practice.

Core antibiotics in respiratory disease

AntibioticRouteTypical adult doseCommon respiratory indicationsImplications for physiotherapy
AmoxicillinOral500 mg–1 g tdsMild CAP, pneumococcal pneumonia, AECOPD, otitis, sinusitisFirst-line for most community-acquired infection in non-penicillin-allergic patients
Amoxicillin-clavulanateOral875/125 mg bdModerate CAP, aspiration pneumonia, bronchiectasis exacerbationBroader gram-negative and anaerobic cover. GI side effects common
Amoxicillin-clavulanateIV1.2 g tdsModerate-severe CAP, aspiration pneumonia, HAP (low risk), bronchiectasis exacerbationIV-to-oral switch typically at 48–72 h
DoxycyclineOral100 mg bdCAP (atypical cover), AECOPD, Mycoplasma, ChlamydiaFirst-line option in penicillin-allergic CAP. Photosensitivity warning
AzithromycinOralAcute: 500 mg day 1 then 250 mg daily ×4.
Maintenance (bronchiectasis, CF): 250–500 mg three times weekly
Atypical CAP, pertussis, Legionella. Long-term low-dose for frequent exacerbatorsLong half-life. QT prolongation risk. Before maintenance therapy is started, sputum must be cultured for NTM — macrolide monotherapy in undiagnosed NTM infection induces resistance and can remove the key drug from a future treatment regimen. Baseline QTc; hearing assessment in long-term use
AzithromycinIV500 mg dailySevere CAP, LegionellaIV reserved for severe disease
ClarithromycinOral500 mg bdAtypical CAP, NTM combination therapyMore drug interactions than azithromycin (CYP3A4 inhibitor)
RoxithromycinOral300 mg daily or 150 mg bdAtypical CAP, AECOPDAustralian macrolide alternative
CefalexinOral500 mg qidStep-down from IV cephalosporinLimited respiratory role as monotherapy
CefuroximeOral / IV500 mg bd PO; 750 mg–1.5 g tds IVCAP, AECOPDLimited atypical cover
CeftriaxoneIV / IM1 g daily (2 g for severe CAP)Moderate-severe CAP, hospitalised pneumonia, empirical sepsisOnce-daily dosing. Pair with macrolide for atypical cover
CefepimeIV1–2 g bd–tdsHAP/VAP, febrile neutropenia, Pseudomonas coverAnti-pseudomonal fourth-generation cephalosporin
Piperacillin-tazobactamIV4.5 g qid (or extended-infusion)HAP, aspiration with severe sepsis, neutropenic fever, CF exacerbation with PseudomonasBroad gram-negative including Pseudomonas + anaerobes
MeropenemIV1 g tds (2 g tds for severe)Severe HAP/VAP, MDR gram-negatives, CF/bronchiectasis with resistant PseudomonasReserved per stewardship
CeftazidimeIV1–2 g tdsPseudomonas in CF, bronchiectasis, HAPAnti-pseudomonal cephalosporin
CiprofloxacinOral / IV500–750 mg bd PO; 400 mg bd–tds IVPseudomonas in bronchiectasis/CF exacerbation, LegionellaTendinopathy risk (especially Achilles, in older patients on concurrent steroids — relevant to exercise programmes), QT prolongation
MoxifloxacinOral / IV400 mg dailySevere CAP, penicillin-allergic CAPQT prolongation, hepatotoxicity, photosensitivity
VancomycinIV25–30 mg/kg loading then 15–20 mg/kg bdMRSA pneumonia, post-influenza S. aureus, severe HAPTrough or AUC monitoring required. Nephrotoxicity
LinezolidOral / IV600 mg bdMRSA or VRE pneumoniaExcellent oral bioavailability. Thrombocytopenia with use >2 weeks
Trimethoprim-sulfamethoxazoleOral / IV160/800 mg bd (PJP treatment up to 5 mg/kg of trimethoprim qid)PJP treatment and prophylaxis, Stenotrophomonas, NocardiaHyperkalaemia, AKI. Drug interactions (warfarin, methotrexate)
MetronidazoleOral / IV400 mg tds PO; 500 mg tds IVAnaerobic cover in aspiration pneumonia, lung abscessAvoid alcohol — disulfiram-like reaction
Tobramycin / gentamicinIV / inhaledIV: weight-based once daily; inhaled tobramycin 300 mg bd (cycle on/off)IV: empirical gram-negative sepsis. Inhaled: chronic Pseudomonas suppression in CFIV: nephrotoxicity, ototoxicity. Inhaled: bronchospasm, voice change
Colistin / colistimethateInhaled / IVInhaled 1–2 MIU bd; IV per protocolPseudomonas eradication in CF/bronchiectasis; MDR gram-negatives IVInhaled: bronchospasm — pre-treat with bronchodilator. IV: nephrotoxicity
AztreonamInhaled / IVInhaled 75 mg tds (cycle on/off); IV 1–2 g tdsInhaled: chronic Pseudomonas suppression in CF. IV: gram-negative in severe penicillin allergyInhaled: cough, wheeze on initiation
Rifampicin (combination)Oral / IV450–600 mg dailyTB combination, NTM combination, persistent MRSAPotent CYP inducer — major drug interactions. Orange discolouration of secretions, urine, contact lenses
Anti-tuberculous (HRZE) combinationOralWeight-based per Australian TB guidelinesActive tuberculosis: 2 months HRZE then 4 months HRManaged through Queensland Health TB services. Multiple toxicities

Physiotherapy timing around IV antibiotics: airway clearance is generally most effective when scheduled 30–60 minutes before a nebulised antibiotic (so the drug is deposited in cleared airways) and not immediately after IV antibiotic infusion if the patient is symptomatic.3

Inhaled antibiotics (e.g. tobramycin, colistin, aztreonam) used long-term in bronchiectasis and cystic fibrosis are covered under nebulised therapy. Always clear the airways first, then give the inhaled antibiotic, and pre-treat with a bronchodilator to reduce bronchospasm.

References & evidence base

  1. Global Initiative for Chronic Obstructive Lung Disease. Global strategy for the diagnosis, management and prevention of COPD: 2026 report. GOLD; 2026. Available at: goldcopd.org
  2. Therapeutic guidelines: antibiotic. Melbourne: Therapeutic Guidelines Limited; updated June 2026. Available at: tg.org.au
  3. Australian Medicines Handbook. Adelaide: Australian Medicines Handbook Pty Ltd, 2026.

References are numbered in citation order (Vancouver/BMJ style) and were current at the time of writing. Guidelines are living documents — verify against the latest version before clinical use. For drug-specific detail, including full product information, consult MedsInfo.

Important: This page is a clinical reference summary, not a prescribing guide or medical advice. All doses are typical adult doses and require individual adjustment; prescribing decisions rest with the treating medical team and current editions of Therapeutic Guidelines / the Australian Medicines Handbook. Always take medicines as prescribed and ask your doctor or pharmacist before changing anything. For personalised assessment, contact Inspire Clinic.

Corrections: If something on this page is wrong, out of date or unclear, we want to know. Email reception@inspireclinic.au with the page name and what you believe is incorrect. Substantive corrections are made promptly, and the guide’s version and last-updated date are changed to reflect it.